Results: The MFA scale was based on the Diamond-Forrester, Duke, CADC models for assessing the risk of coronary artery stenosis, as well as the SMART scale, since endpoint predictors should be associated with atherosclerosis, however, there was only statistical significance of such a relationship for total cholesterol (TC) (r=0.619; p<0.001), low-density lipoproteins (LDL) (r=0.686; p<0.001), gender (r=0.295; p=0.049). The atherogenic coefficient (CA), which is not included in these scales, had a fairly high correlation coefficient r=0.484 (p=0.048). The degree of interaction of factors and multifocal atherosclerosis was as follows. For TC, the influence of the factor on the development of multifocal atherosclerosis was 3.731 at p=0.040, while another point of view was 34.517 at p<0.001; thus, leading to a ratio of 1:9.25; the same pattern is typical for LDL - Factor ? MFA - 4.798 at p=0.032, while MFA ? factor 24.731 at p<0.001. The same pattern was observed for CA: 4.123 p=0.048 and 4.587 at p=0.023. The development of MFA with bronchogenic lung cancer had an effect 9.25 times higher on the level of TC in the blood, compared with the effect of total cholesterol on the development of multifocal atherosclerosis, the same is true for LDL (5.15 times). The level of CA had a interaction of 1:1.112. The level of high density lipoproteins did not affect the development of MFA (F=1.80, p=0.19). In patients with MFA, the male predominated (F=3.973, p=0.048). This model was tested using ROC analysis: the area under the curve was 0.798, which corresponded to a “good” score on the area under the curve scale.

1) Multifocal atherosclerosis in a patient with bronchogenic lung cancer is a factor that increases blood levels of TC (9.25:1) and LDL (5.15:1);

2) Male with bronchogenic lung cancer have a higher risk of development MFA than female (4.021:1).